Project 170693

SOCS3 regulation of insulin and leptin sensitivity in obesity

170693

SOCS3 regulation of insulin and leptin sensitivity in obesity

$309,327
Project Information
Study Type: Other Mechanistic_Study
Therapeutic Area: Endocrinology
Research Theme: Biomedical
Disease Area: obesity, metabolic syndrome, diabetes
Data Type: Canadian
Institution & Funding
Principal Investigator(s): Steinberg, Gregory R
Institution: McMaster University
CIHR Institute: Nutrition, Metabolism and Diabetes
Program: Operating Grant
Peer Review Committee: Endocrinology
Competition Year: 2008
Term: 3 yrs 0 mth
Abstract Summary

Canada is experiencing an epidemic of obesity that is contributing to diabetes, heart disease, and premature death. This proposal investigates why being obese causes the metabolic syndrome with a specific emphasis on how muscle and fat respond to insulin. An improved understanding about how hormones regulates the body's storage and breakdown of fat and responsiveness to insulin will enable the development of new medicines for the treatment of obesity and the prevention of diabetes.

Research Characteristics

This project includes the following research characteristics:

Regulatory Pathway
Comorbidity Focus
Knowledge Translation Focus
Biomarker Endpoints
Study Justification

"investigates why being obese causes the metabolic syndrome with a specific emphasis on how muscle and fat respond to insulin"

Novelty Statement

"An improved understanding about how hormones regulates the body's storage and breakdown of fat and responsiveness to insulin will enable the development of new medicines for the treatment of obesity and the prevention of diabetes."

Methodology Innovation

investigating SOCS3 regulation of insulin and leptin sensitivity in obesity

Keywords
Amp-Activated Protein Kinase Insulin Resistance Leptin Resistance Metabolism Obesity Suppressor Of Cytokine Signaling